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  1. Pubblicazioni

ONCOGENE

Rivista
Codice:
E125368
ISSN:
0950-9232
  • Dati Generali

Dati Generali

Pubblicazioni (16)

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A KRAS variant is a biomarker of poor outcome, platinum chemotherapy resistance and a potential target for therapy in ovarian cancer.
Articolo
Activation of diacylglycerol kinase alpha is required for VEGF-induced angiogenic signaling in vitro.
Articolo
Biological activity of substrate-bound basic fibroblast growth factor (FGF2): recruitment of FGF receptor-1 in endothelial cell adhesion contacts.
Articolo
Contribution of vascular endothelial growth factor receptor-2 sialylation to the process of angiogenesis
Articolo
Deregulated FGFR3 mutants in multiple myeloma cell lines with t(4;14): comparative analysis of Y373C, K650E and the novel G384D mutations.
Articolo
Detection and subcellular localization of an AML1 chimeric protein in the t(8;21) positive acute myeloid leukemia.
Articolo
FGF2-induced upregulation of DNA polymerase-delta p12 subunit in endothelial cells.
Articolo
Fibroblast-derived prolargin is a tumor suppressor in hepatocellular carcinoma
Articolo
Gene expression profile in fibroblast growth factor 2-transformed endothelial cells.
Articolo
HDAC7 inhibition resets STAT3 tumorigenic activity in human glioblastoma independently of EGFR and PTEN: new opportunities for selected targeted therapies
Articolo
HIV Tat protein has the properties of a heparin-binding angiogenic growth factor
Articolo
Paracrine and autocrine effects of fibroblast growth factor-4 in endothelial cells.
Articolo
Sequential analysis of multistage hepatocarcinogenesis reveals that miR100 and PLK1 dysregulation is an early event maintained along tumor progression.
Articolo
Southern Europe as an example of interaction between various environmental factors. A systematic review of the epidemiologic evidence.
Articolo
Syndecan-1 increases B-lymphoid cell extravasation in response to HIV-1 Tat via αvβ3/pp60src/pp125FAK pathway
Articolo
The transcriptional corepressor MTG16a contains a novel nucleolar targeting sequence deranged in t (16; 21)-positive myeloid malignancies.
Articolo
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